HIGHLIGHTS
- who: MCAK and collaborators from the School of Life Sciences, University of Nottingham, Medical School, QMC, Nottingham , UH, UK have published the Article: The family-specific α4-helix of the kinesin-13, MCAK, is critical to microtubule end recognition, in the Journal: (JOURNAL) of 08/07/2022
- what: The authors show that the mutated residues, which are in the C-terminal half of the a4-helix, are not important for either microtubule or unpolymerized tubulin activation of the ATP cleavage step of ATP turnover by MCAK. It is clear from the work described here that . . .
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