Sod2 promotes gastric tumorigenesis mediated by helicobacter pylori and enhances resistance to 5-fluorouracil in gastric cancer

HIGHLIGHTS

  • What: The authors aimed to investigate the biological characteristics and function of SOD2 in response to H. pylori infection in GC. The aim of this study was to explore whether H. pylori infection could promote SOD2 expression, further facilitating gastric carcinogenesis.
  • Who: Posted Date November and collaborators from the Naval Medical University have published the research: SOD2 promotes gastric tumorigenesis mediated by Helicobacter pylori and enhances resistance to 5- fluorouracil in gastric cancer, in the Journal: (JOURNAL)
  • How: The final concentrations used in the experiments were as follows ferrostatin-1 2 µM and . . .

     

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