HIGHLIGHTS
- who: Hugh from the DepartmentWashington University Medicine, StLouis, Missouri, USA have published the Article: 07_pri4 new, in the Journal: (JOURNAL)
- what: The application of a cell biological approach to the study of prion diseases has allowed us to define the normal trafficking pathways followed by PrPC, and to develop a cell culture system for the de novo formation of PrPSc from PrP molecules carrying pathogenic mutations.
- how: This result was not unexpected since an equivalent mutation in the second glycosylation consensus site of human PrP (T183A) has recently been associated with a familial . . .
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