HIGHLIGHTS
SUMMARY
Cardiovascular diseases are significant reasons of morbidity and mortality in obese individuals. In obesity, increased free fatty_acids can cause mitochondrial dysfunction and free oxygen radical formation and eventually endothelial dysfunction. As a result of increased free fatty_acids and accompanying activated inflammatory pathways, insulin resistance and oxidative stress may be generated. Obesity leads to malfunction in particularly visceral type of adipose_tissue. As a result of cytokines originating from adipose_tissue omentin levels decrease in obesity. Similarly, the NO levels overweight/obese midlevel intensity exercise program resulted in subjects did not increase significantly related to enhanced endothelial . . .
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