Oxidative stress and nuclear reprogramming: a pilot study of the effects of reactive oxygen species on architectural and epigenetic landscapes

HIGHLIGHTS

SUMMARY

    Overall, ROS may contribute to the pathogenesis through both genetic and epigenetic mechanisms, for example by altered gene_expression modulation due to hypermethylation or hypomethylation. One of the main gene_expression regulation mechanisms is represented by DNA methylation. According to the most diffused interpretation, DNA methylation in the form of 5-methylcytosine (5mC) is a hallmark of gene repression, by altering the binding affinity of transcription factors and/or affecting chromatin structure, reducing nucleosome flexibility, and determining a more compact environment, which in turn negatively modulates gene_expression. 5hmC is the first and most stable intermediate in . . .

     

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