HIGHLIGHTS
SUMMARY
As another HHCys-related effect, reduction in angiogenesis results from the downregulation of pro-angiogenic factors in serum (FAS, S100A4, TNFRSF12A, and TGFBR3; Wiley et_al, 2001; Lambert et_al, 2003; Ambartsumian et_al, 2019; Wang et_al, 2019) and CSF (DLK1 and ACVRL1; Urness et_al, 2000; Huang et_al, 2018), which might display an interesting mechanistic aspect in terms of vascular contribution to cognitive decline and dementia. For instance, downregulation of TNFRSF11B, TGFBR3, FSTL3, and WISP1 and upregulation of IL1a in serum are associated with diminished bone mineralization, decreased bone anabolism, or increased bone catabolism (Lee et_al, 2002 . . .
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