HIGHLIGHTS
- who: Turati et_al Experimental Hematology and collaborators from the (UNIVERSITY) have published the article: CORRESPONDENCE Open Access, in the Journal: (JOURNAL)
SUMMARY
I this context, the lymphangiogenic switch is induced and sustained by tumor-derived pro-lymphangiogenic factors, including the Vascular Endothelial Growth_Factor family members VEGF-C and VEGF-A, nevertheless scattered pieces of evidence indicate that also the pro-angiogenic Fibroblast Growth_Factor-2 (FGF2) may play a relevant role in activating lymphangiogenesis through different mechanisms. It has been shown that the combination of VEGF-A, sphingosine-1-phosphate (S1P) and FGF2 represents . . .
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