H3k4me3 mediates uterine leiomyoma pathogenesis via neuronal processes, synapsis components, proliferation, and wnt/β-catenin and tgf-β pathways

HIGHLIGHTS

• who: Maru00eda Cristina Carbajo-Garcu00eda from the (UNIVERSITY) have published the research work: H3K4me3 mediates uterine leiomyoma pathogenesis via neuronal processes, synapsis components, proliferation, and Wnt/u03b2-catenin and TGF-u03b2 pathways, in the Journal: (JOURNAL)
• what: The authors aimed to identify if the H3K4me3 regulates the of genes involved in pathogenesis. The authors aimed to describe whether the histone mark H3K4me3 regulates key pathways and driver genes involved in UL pathogenesis, through an in-depth integrative analysis of the transcriptome and H3K4me3 CHIP-seq profiles in ULs compared to their adjacent MM, regardless their . . .
  

   

  

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