HIGHLIGHTS
SUMMARY
Type 1 diabetes (T1D) is a multifactorial disease characterized by a progressive destruction of pancreatic β cells, triggered by an autoimmune_response, that leads to a lack of insulin production. After the hypothesis was formulated at the end of the eighties, gut microbial markers associated with insulin resistance and evidence of the role of gut microbiota (GM) in the immune-mediated onset of T1D were observed in rodent models. Among the main aims of this work, to unveil the differential regulation of microbial proteins in T1Ds, compared with healthy CTRLs and SIBLs, was important to individuate . . .
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