HIGHLIGHTS
- who: April and collaborators from the University Hospital La Paz, Spain have published the research work: The mechanism of chronic intracellular infection with Brucella spp, in the Journal: (JOURNAL)
- what: This study provides a new explanation for the ability of B. abortus to grow and replicate in macrophages in the early stages of infection (Wei et_al, 2015).
SUMMARY
Since Brucella hardly activates the complement system through the classical pathway or activates granulocytes, it causes very little tissue damage and does not cause obvious blood changes, such as leukocytosis, increased neutrophilia, and . . .
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