Long qtc in hypertrophic cardiomyopathy: a consequence of structural myocardial damage or a distinct genetic disease?

HIGHLIGHTS

• who: Francesco Cava and colleagues from the For this study we considered a prospectively collected database, including , consecutive HCM outpatients carrying pathogenic variants in sarcomere genes, based on previous genetic screenings, referred to the tertiary HCM Center of Sant`Andrea Hospital, Sapienza University of Rome between January , and June Each patient was evaluated considering the HCM echocardiographic findings detected by transthoracic Doppler echocardiography (apical, septal hypertrophy), the electrocardiographic findings, the clinical history and New York Heart Association (NYHA) classification, the pedigree analysis. The following echocardiographic parameters were considered: Left Ventricular (LV) end-diastolic diameter (LVEDd, parasternal long . . .
  

   

  

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