HIGHLIGHTS
SUMMARY
The underlying genomic basis for host resistance to infectious diseases is predominantly caused by polygenic traits, where many genes or variants collectively contribute to the phenotype. Knowledge of their high level of plague resistance has existed since the early 1950s and individual hetero- a 2 | geneity in response to infection has been revealed by challenge experiments. Although some of these studies were unable to determine candidate genes due to the low resolution of their method, others identified multiple candidate genes involved in immune-related processes as well as genes lacking a known immune function . . .
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