HIGHLIGHTS
SUMMARY
Although the detailed molecular mechanism remains imperfectly understood, the authors conclude that PTH-AS may contribute to tumor development, possibly through IRDS gene upregulation. lncRNAs regulate tissue-specific gene_expression through interaction with DNA, proteins, and RNA, thereby driving vital processes, such as differentiation, development, and immunoregulation. The authors describe the characteristics of this lncRNA and the effect of forced expression on PTH-nonexpressing human breast cancer T47D cells on global gene_expression including the PTH gene by microarray analysis. Ectopic PTH expression cannot be induced by PTH-AS expression alone PTH-AS expression was detected . . .
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