Altered cortical synaptic lipid signaling leads to intermediate phenotypes of mental disorders

HIGHLIGHTS

• What: This analysis provides complete distributions of credible values for group means and their differences . These findings are well in line with the idea that higher activation in 3550 memory related regions represent a compensatory activity aiming to rescue cognitive performance. One limitation of this study is the relatively small human subjects sample, which may result from the low frequency of the analyzed PRG-1 SNP (0,87%). This study provides a mechanistic chain reaching from molecular to phenotypic alterations as one example of the longsought causes of E/I imbalance .
• Who: Oliver Tüscher . . .
  

   

  

  If you want to have access to all the content you need to log in!

  Thanks :)

  Username or E-mail:

  If you don't have an account, you can create one here.