HIGHLIGHTS
SUMMARY
In 1994, Aitken proposed the loss of sperm function subsequent to the induction of peroxidative damage by ROS originating from the spermatozoa as a theory for male infertility. Before further exploring the pathophysiological mechanisms behind the relationship between OS and male infertility, it is imperative to properly characterise the oxidant and antioxidant elements playing in the particular setting of human spermatozoa. The author propounds that, under in_vivo conditions, the induction of OS on spermatozoa by infiltrating leukocytes is unlikely given the protective properties of seminal plasma. In recent work Malmir and colleagues studied the . . .
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