Association of atp6v1b2 rs1106634 with lifetime risk of depression and hippocampal neurocognitive deficits: possible novel mechanisms in the etiopathology of depression

HIGHLIGHTS

• who: X Gonda from the polymorphisms implicated in depression GWAS-s, even if only with a weak effect, with respect to their possible role and mechanismCONFLICT OF INTEREST JFWD variously performed consultancy, speaking engagements and research for Bristol-Myers Squibb, AstraZeneca, Eli Lilly, Schering Plough, Janssen-Cilag and Servier (all fees are paid to the University of Manchester to reimburse them for the time taken) have published the research work: Association of ATP6V1B2 rs1106634 with lifetime risk of depression and hippocampal neurocognitive deficits: possible novel mechanisms in the etiopathology of depression, in the Journal: (JOURNAL)
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