HIGHLIGHTS
- who: Andreas Franz and colleagues from the (UNIVERSITY) have published the paper: Branch point strength controls species-speci fi c CAMK2B alternative splicing and regulates LTP, in the Journal: (JOURNAL)
- what: The authors report species-specific alternative splicing of CAMK2B and identify several primate-specific splice isoforms, which are generated through exclusion of exon 16. Using RNA-Seq and minigene analyses, the authors show that weakening of the branch point (BP) sequence during evolution directs primate-specific exon 16 exclusion. Focusing on CAMK2B, the authors show that the primate-specific protein isoforms reach a higher . . .
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