HIGHLIGHTS
SUMMARY
Stroke is one of the most frequent neurological disorders leading to chronic disability and is recognized as a major cause of morbidity and mortality worldwide. The type III deiodinase, D3, barely expressed in adult tissues, is the main THs inactivator, prompting the conversion of both T4 and T3 in THs` inactive forms. In the attempt to shed light on the involvement of THs in modulating the different aspects of ischemic diseases and the functional recovery of patients affected by this pathology, this narrative review aims to describe the role exerted by THs in the . . .
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