HIGHLIGHTS
SUMMARY
Different cell processesand accounting cancer cell proliferation and progression, probablyfor responsible triple-negative breast cancer cell proliferation and progression, and probably responsible for chemoresistance, including autophagic/apoptotic cell death, oxidative stress, genomic for chemoresistance, including autophagic/apoptotic cell death, oxidative stress, genomic cytoproinstability, cell cycle progression and cell migration, were evaluated. The genetic instability and deregulation of cell cycle checkpoints are known to favor the transformation processes, allowing the proliferation of altered cells under conditions inadequate for normal cells. This evidence suggests that the cell cycle arrest induced by the sesquiterpene in the MDA . . .
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