Cigarette smoke extract-mediated fabp4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 mapk/mk2 signaling pathway

HIGHLIGHTS

• who: Wei Zhang from the District, Hangzhou , Zhejiang, China have published the article: Cigarette smoke extract-mediated FABP4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 MAPK/MK2 signaling pathway, in the Journal: (JOURNAL)
• what: The authors aim to explore the potential link between
• how: For further study the authors utilized 2% CSE to treat 16HBE cells for 48 h to perform the subsequent experiments. shRNAu2011mediated FABP4 abrogation alleviates CSEu2011mediated 16HBE cell viability decline and apoptosis To investigate the role of FABP4 in 16HBE . . .
  

   

  

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