HIGHLIGHTS
SUMMARY
From the perspective of plant-virus coevolution, systemic necrosis can be hardly viewed as selectively advantageous for the individual plant because, at odds with full resistance, fitness may drop to zero upon early infection. One such phenotype is virus-induced plant systemic necrosis, which does not localize virus infection and results in the death of the infected plant. A reduction in virus multiplication associated with systemic necrosis has also been reported in Arabidopsis plants with the TuNI gene that determines systemic necrosis in response to infection by turnip mosaic virus (TuMV), where virus accumulation . . .
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