HIGHLIGHTS
- who: Marion Laudette from the Animals Mice were housed in a pathogen-free facility and all animal experiments were approved by the Animal Care and Use Committees of the University of ToulouseEpac1-deficient mice (Epac1-/-) have been engineered in our laboratory as previously described [43]. have published the paper: Cyclic AMP-binding protein Epac1 acts as a metabolic sensor to promote cardiomyocyte lipotoxicity, in the Journal: (JOURNAL) of 29/03/2021
- what: The authors report that a diet-dependent signal, palmitate regulates the intracellular synthesis of cAMP to activate Epac1, which in turn promotes lipid . . .
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