HIGHLIGHTS
- who: Office 2004 Test Drive User from the Comprehensive Cancer Center, Buffalo, NY, USA have published the paper: dSir2 and Dmp53 interact to mediate aspects of CR-dependent life span extension in D, in the Journal: (JOURNAL)
SUMMARY
As suggested in 2006, by slowing down the mTORdriven developmental program, rapamycin must slow down quasi-programmed aging. Targeting development with rapamycin must lead to a longer lifespan. According to hyper-function theory, aging is a quasiprogram, a purposeless continuation of the growth program that has not been switched off upon its completion. Aging is . . .
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