Enhanced th17-cell responses render ccr2-deficient mice more susceptible for autoimmune arthritis

HIGHLIGHTS

• who: Rishi R. Rampersad and colleagues from the Department of Medicine, Thurston Arthritis Research Center, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, United States of America, Division have published the paper: Enhanced Th17-Cell Responses Render CCR2-Deficient Mice More Susceptible for Autoimmune Arthritis, in the Journal: (JOURNAL) of May/14,/2011
• what: The aim of a second booster is to increase the overall disease severity so that the difference between control mice and gene-deficient or treatment animals that have less disease can be apparent. The authors show that Th17 . . .
  

   

  

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