Er-stress-induced secretion of circulating glucose-regulated protein 78kda (grp78) ameliorates pulmonary artery smooth muscle cell remodelling

HIGHLIGHTS

  • who: Muntadher Al Zaidi from the Heart Center Bonn, Department of Internal Medicine II, University Hospital Bonn, Venusbergu2011Campus, Bonn, Germany have published the research work: ER-stress-induced secretion of circulating glucose-regulated protein 78kDa (GRP78) ameliorates pulmonary artery smooth muscle cell remodelling, in the Journal: (JOURNAL)
  • what: The aim of this study is to investigate its significance in PAH.
  • how: For risk stratification the authors used the number of low-risk criteria present to stratify patients.

SUMMARY

    Administration of GRP78 was found to resolve inflammation in mononuclear cells . . .

     

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