Exogenous 3-iodothyronamine (t1am) can affect phosphorylation of proteins involved on signal transduction pathways in in vitro models of brain cell lines, but these effects are not strengthened by its catabolite, 3-iodothyroacetic acid (ta1)

HIGHLIGHTS

• who: Lavinia Bandini et al. from the Department of Pathology, University of Pisa, Pisa, Italy have published the research: Exogenous 3-Iodothyronamine (T1AM) Can Affect Phosphorylation of Proteins Involved on Signal Transduction Pathways in In Vitro Models of Brain Cell Lines, but These Effects Are Not Strengthened by Its Catabolite, 3-Iodothyroacetic Acid (TA1), in the Journal: Life 2022, 12, x FOR PEER REVIEW of /2022/
• what: The authors compared the effects of and on protein belonging to signal transduction pathways assuming that may strengthen AM's effects in brain tissue. In the experiments aimed . . .
  

   

  

  If you want to have access to all the content you need to log in!

  Thanks :)

  Username or E-mail:

  If you don't have an account, you can create one here.