Experimental atopic dermatitis depends on il-33r signaling via myd88 in dendritic cells

HIGHLIGHTS

  • who: Changwei Li from the Experimental and Molecular Immunology and Neurogenetics (INEM), UMR , CNRS and University of of Life Sciences, East China have published the research work: Experimental atopic dermatitis depends on IL-33R signaling via MyD88 in dendritic cells, in the Journal: (JOURNAL)
  • what: The authors report a critical role of IL-33/ST2 signaling as experimental AD in IL-33- and ST2- / - mice was drastically reduced with only minor Cell Death and Disease reduction in IL-1αβ- / - mice, but no difference in IL-36 R- / - and IL-36Ra- / - mice. The authors report that . . .

     

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