Glutamatergic dysfunction leads to a hyper-dopaminergic phenotype through deficits in short-term habituation: a mechanism for aberrant salience

HIGHLIGHTS

• who: Marios C. Panayi from the Genetic construction, breeding, and genotyping of Gria1-/- mice was as previously described [28]Briefly, homozygous knockout (Gria1-/-) and wild-type (WT) littermates were generated from heterozygousheterozygous breeding pairs. All procedures were performed in accordance with the United Kingdom Animals (Scientific Procedures) Act of , and were approved by the University of Oxford Ethical Review Board. Subjects for anaesthetized voltammetry recordings were , WT mice (Male n=3) and , Gria1-/- mice (Male n=3). During freely moving in-vivo voltammetry, data were obtained from N=, working electrodes (WT n=16, Gria1-/- n=9) targeting . . .
  

   

  

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