HIGHLIGHTS
- What: The authors propose that expression of MST2∆7 could provide advantages to abnormal cells, contributing to the onset of cancer, as resistance to cell death and incapability of cells to become/stay quiescent disrupts cell number homeostasis leading tumor growth31,32.
- Who: Posted Date January et al. from the University of Sao Paulo https://orcidorg/0000-0003-3475, have published the article: Identification of a novel alternative splicing isoform of the Hippo kinase STK3/MST2 with impaired tumor suppressor activities, in the Journal: (JOURNAL)
- How: For such the authors used the S1 . . .
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