Identifying foetal forebrain interneurons as a target for monogenic autism risk factors and the polygenic 16p11.2 microdeletion

HIGHLIGHTS

• who: Yifei Yang from the All rats were bred in-house according to Home Office UK legislation and licenses approved by the University of Edinburgh Ethical Review Committees and Home OfficeAnimal husbandry was in accordance with UK Animals (Scientific Procedures) Act of , regulations. Rat, p11., DEL rat model (16p11.2+/-) was generated by CRISPR/Cas, genome editing of the Sprague Dawley line [59]. Rats were maintained on the Sprague Dawley background and genotyped by PCR as previously described [59]. , rat tissue was fixed by transcardial perfusion with, % paraformaldehyde in PBS, brains were then dissected and immersed in . . .
  

   

  

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