HIGHLIGHTS
SUMMARY
The observation of a close similarity between growth factor receptors endowed with a tyrosine kinase enzymatic activity and virally encoded oncogenes introduced the general idea that gain of function somatic mutations in genes coding for endogenous kinases involved in the regulation of cell proliferation may be responsible of the neoplastic phenotype. Motile cilia are found on respiratory epithelial_cells, be directly involved in the Wnt or Shh pathway. In experiments using an epithelial_cell line of the choroid plexus in culture, TRPV4 (transient receptor potential channel 4) was identified as a channel protein which, once activated . . .
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