HIGHLIGHTS
- who: ROS et al. from the Jinan University, China have published the research work: Malvidin alleviates mitochondrial dysfunction and ROS accumulation through activating AMPK- /UCP2 axis, thereby resisting in ammation and apoptosis in SAE mice, in the Journal: (JOURNAL)
- what: The aim of this study is to investigate the pathogenesis of SAE and look for effective prophylactic drugs aiming to provide early clinical intervention for SAE patients to treat brain dysfunction, thereby reducing sepsis mortality and improving the quality of life of SAE patients. This study systematically elucidated the molecular mechanisms of SAE from the . . .
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