HIGHLIGHTS
SUMMARY
Several studies have revealed m6 A`s involvement in regulating the formation and consolidation of memory, and abnormality of m6 A is involved in AD, but the specific functions of m6 A modification in the process of memory remain to be illustrated (Zhang et_al, 2018; Huang et_al, 2020; Shafik et_al, 2021; Zhao et_al, 2021). Studies have reported that the deficiency of IEGs represents common epigenetic changes observed in AD models, which indicates that IEGs contribute to the pathogenesis of AD (Anderson et_al, 1994; Dickey et_al, 2004). It has been reported that the newly synthesized . . .
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