HIGHLIGHTS
- who: Jingli Cai from the (UNIVERSITY) have published the article: METTL3 promotes glycolysis and cholangiocarcinoma progression by mediating the m6A modification of AKR1B10, in the Journal: (JOURNAL)
- what: The authors aimed to excavate the biological role and mechanism of the m6A methyltransferase in_(CCA). The authors intended to reveal the biological role of m6A modification of aldo-keto reductase family 1 member B10 (AKR1B10) mediated by METTL3 in CCA progression. The study proposed that METTL3 may be a target of potential inhibitors for blocking glycolysis for application in CCA therapy. Given that METTL3 promotes the . . .
If you want to have access to all the content you need to log in!
Thanks :)
If you don't have an account, you can create one here.