HIGHLIGHTS
- who: Calvin Wong et al. from the DepartmentMcGill University, Boston, Masschusetts, USA have published the research work: mTORC2 mediates structural plasticity in distal nociceptive endings that contributes to pain hypersensitivity following inflammation, in the Journal: (JOURNAL)
- what: The authors investigated whether mTORC2 plays a role in the development of pain hypersensitivity in response to tissue inflammation. The authors demonstrate that mTORC2 activation is an important driver of inflammatory pain and propose that mTORC2-dependent hypersensitivity is mediated via structural changes in nociceptive free nerve endings. To study the mechanism by which activation of mTORC2 promotes . . .
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