HIGHLIGHTS
SUMMARY
AML is featured with the infiltration of malignant myeloid progenitor cells into the peripheral blood, bone marrow, and other tissues, which often progress rapidly leading to poor prognosis (De Kouchkovsky and Abdul-Hay, 2016; Papaemmanuil et_al, 2016). Numerous studies have described that methyltransferases ("writers"), demethylases ("erasers"), and the common m6A binding protein ("reader") were significantly upregulated in different subtypes of AML. Microarray data GSE37642 (Li et_al, 2013; Herold et_al, 2014; Kuett et_al, 2015; Herold et_al, 2018) was extracted from the Gene Expression Omnibus (GEO) database (http://www.ncbi.nlm.nih. gov/geo). A lot . . .
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