HIGHLIGHTS
- who: Hendrikus J. Van Heesbeen and collaborators from the Swammerdam Institute for Life Sciences, University of Amsterdam, Science Park, Room, XH Amsterdam, The Netherlands have published the Article: Neuronal Dot1l Activity Acts as a Mitochondrial Gene-Repressor Associated with Human Brain Agingvia H3K79 Hypermethylation, in the Journal: (JOURNAL)
- what: The authors show in mouse models that in post-mitotic neurons the turnover of H3K79 methylation is very high, resulting in a major loss of the methylation marks (H3K79Me2/Me1) within 48 h. If the authors compare the rapid loss of post-mitotic neuronal H3K79me2 following . . .
If you want to have access to all the content you need to log in!
Thanks :)
If you don't have an account, you can create one here.