HIGHLIGHTS
SUMMARY
Interstitial pneumonia due to SARS-CoV-2 can be complicated by possible neurological involvement with mechanisms of direct CNS infection and/or with para-infective mechanisms of the peripheral nervous system (PNS), shown by some neuropathological findings of COVID-19 patients, as recently reviewed. In other, larger series of patients with GBS associated with COVID-19, emerged a higher frequency of subjects in which GBS started while COVID-19 symptoms were still ongoing. COVIDGBS patients had respiratory symptoms at presentation to the ED, and the length of these symptoms was significantly longer than in . . .
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