HIGHLIGHTS
- who: January and collaborators from the University of Calgary, Adjunct Associate Professor, Canada have published the article: Neutralization of excessive CCL28 improves wound healing in diabetic mice, in the Journal: (JOURNAL)
- what: The authors investigate a mechanism by which an excessive level of chemokine CCL28, through its receptor CCR10, impairs wound healing in patients and mice with T2DM. The authors propose that neutralization of excessive CCL28 with 1 u03bcg anti-CCL28 Ab reduces and perhaps normalizes CCL28 signaling in endothelial and non-endothelial CCR10 expressing cells resulting in reduced inflammation and eNOS downregulation, thereby improving . . .
If you want to have access to all the content you need to log in!
Thanks :)
If you don't have an account, you can create one here.