HIGHLIGHTS
- who: from the approved by the Stanford University Institutional Review BoardTumor samples were obtained from patients with pancreatic cancer following surgical have published the paper: OPEN ACCESS, in the Journal: (JOURNAL) of 22/04/2023
- what: The authors show that the inhibition of complement C3a enhances NK cell infiltration in preclinical models of pancreatic cancer, resulting in tumor growth delay. The binding of C3a to its receptor, C3aR on NK cells, leads to the interaction of the C3aR with the integrin lymphocyte function-associated antigen 1 (LFA-1), Herein, the authors provide evidence that NK . . .
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