HIGHLIGHTS
SUMMARY
The hallmark of most neuromuscular diseases is muscle weakness, which often emerges because of motor neuron loss (Garg et_al, 2017). To compensate for this motor neuron loss, collateral sprouting from neighboring motor neurons arises and leads to a re-innervation of muscle fibers, ultimately increasing the size of individual motor units (Edds, 1953; Kugelberg et_al, 1970; Milner-Brown et_al, 1974; Zochodne, 2012). At the same time, the interference signal also shows a high amplitude, since the remaining compensatory oversized motor units innervate a larger number of muscle fibers (i.e., a larger signal source . . .
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