HIGHLIGHTS
- who: BMC Rheumatology et al. from the Johns Hopkins University, Baltimore, MD, United States have published the research work: Oral Abstracts of scientific contributions to GCOM, in the Journal: (JOURNAL)
- what: An alternative explanation for its refractoriness may come from the inability of conventional immunosuppressive therapies to address the specific nature of the T cell autoimmunity present in IBM Here the authors report on translational studies of IBM patient muscle (N=39) and blood samples (N=12) compared with large numbers (N=299) of samples from other muscle diseases identifying a unique cytotoxic lymphocyte signature . . .
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