HIGHLIGHTS
SUMMARY
An early study conducted by Gross et_al, demonstrated that mice depleted of complement by using cobra venom factor were more susceptible to P. aeruginosa pneumonia than untreated mice. In a first study, Mller-Kristensen et_al, used a murine model of burn infection to demonstrate that LP plays a critical role in containing and preventing a systemic spread. Conversely, Kenawy et_al, observed that MASP-2-deficient mice did not display a higher mortality rate than healthy mice in a model of acute lung infection by P. aeruginosa. Mishra et_al, proved that an OprF-deficient P . . .
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