HIGHLIGHTS
SUMMARY
The authors found that several splicing factors are responsible for the alternative splicing of Ki67 exon 7, and SRSF3 can promote exon 7 inclusion. The authors explored the regulatory mechanisms of alternative splicing of Ki67 exon 7, especially the key motif for exon 7 inclusion. Pan-cancer analysis showed that the alternative splicing of Ki67 exon 7 could be widely detected in tumor tissues and normal tissues in 2023, 24, 3872 3 of 23 3 of 20 exon 7 may represent a more attractive potential prognostic marker than the expression levels of total Ki67 . . .
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