SUMMARY
Persons with Parkinson`s disease (PD) demonstrate gait disturbances that usually worsen with disease progression. It has also been hypothesized that the automaticity of movement mediated by the crosstalk of basal ganglia (BG) inputs from the motor, cognitive, and limbic cortical areas, which regularly complement each other in unafected persons, is disrupted in PD with FoG. To support this hypothesis, clear biomarkers are needed that are associated with active gait and the physiological response from neural activity in preparation for and during active gait. Although EEG does not provide the high spatial resolution of other imaging modalities . . .
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