SUMMARY
The majority of plasma-free fatty_acid (FFA) is carried on albumin and provides ATP for the kidney by the mitochondrial β-oxidation of FFA. Almost all plasma fatty_acids are bound to albumin, with only a very small percentage being FFA. According to the energy demand of cells, intracellular fatty_acids are directed to mitochondrial β-oxidation, or stored as triglyceride in intracellular lipid droplets if excessive. The albumin-bound nonesterified fatty_acids (NEFA, also known as FFA) that passed through the damaged glomerular filtration barrier can also be reabsorbed by the proximal tubule through fatty_acid transporter-2 (FATP2), causing lipid accumulation . . .
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