HIGHLIGHTS
SUMMARY
Special emphasis is given to the effects of Oxt on skeletal muscle and to the recent paradigm shifting hypothesis that Oxt may increase the tone of the slow-twitch muscle upregulating Oxt receptor (Oxtr) and triggering the "oxytonic contraction" after cold stress (CS) challenge in mice. Under the regulation of these extracellular and intracellular factors, Oxt neuronal activity and Oxt secretion can meet the demands of body activities in response to environmental changes. It will be important to determine if mice with global loss of Oxt/Oxtrs have impairment in both BAT thermogenesis and . . .
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