HIGHLIGHTS
SUMMARY
This review will summarize the role of the NLRP3 inflammasome complex in the development of CA-CRC and summarize studies that report on the downregulation of NLRP3 activity, to develop therapeutic strategies in IBD as well as in CA-CRC. This review intended to gather all of the recent evidence regarding the role of NLRP3 in the pathogenesis of CA-CRC, as well as the pathways of activation or inactivation. The majority of the studies were experimental studies focusing on three major aspects of the review: means of upregulation of NLRP3 complex, means of . . .
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