HIGHLIGHTS
SUMMARY
Mdx mice, like humans, lack dystrophin, resulting in muscle degradation and regeneration failure, and can be used as an animal model of DMD to assess the effects of various therapies on muscle malnutrition (Zhang and Li, 2018; Radley-Crabb et_al, 2014). Other cell therapies and drug treatments aim to improve muscle function and quality by addressing the pathogenesis and symptoms of DMD (Liu et_al, 2018; Verhaart and Aartsma-Rus, 2019; Zhang et_al, 2019). There are still many disagreements among DMD patients about whether or not to exercise, how to exercise, when to exercise, the . . .
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