The pathobiology of interleukin 11 in mammalian disease is likely explained by its essential evolutionary role for fin regeneration

HIGHLIGHTS

  • who: Stuart A. Cook from the Cardiovascular and Metabolic Disorders Program, Duke-National University of Singapore Medical School, Singapore, Singapore have published the Article: The Pathobiology of Interleukin 11 in Mammalian Disease is Likely Explained by its Essential Evolutionary Role for Fin Regeneration, in the Journal: (JOURNAL)

SUMMARY

    Cardiovascular and Metabolic Disorders Program, Duke-National University of Singapore Medical School, Singapore, Singapore National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore, Singapore GSK3β leading to an mTOR- and SNAI1-driven program of epithelial-to-mesenchymal transition (EMT). This causes epithelial_cell dedifferentiation and . . .

     

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